NOT KNOWN DETAILS ABOUT LDN-27219 PRECLINICAL RESEARCH

In contrast to neurodegerative ailments and cancer, the contribution of TG2 activity to celiac sprue pathogenesis has become rather perfectly founded. The deamidation of unique glutamine residues qualified by TG2 in gluten peptides has long been revealed to enhance peptide binding for the condition-connected proteins HLA-DQ2/8, which can be the onl